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AETIOLOGY
Low back pain is an integral part of
most human lives and causes different degrees of suffering and
disability. The exact cause of pain cannot be identified in most
instances. Approximately 90% of back pain have no identifiable
cause.
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Table 1:
Aetiology of low back pain |
|
Mechanical |
Inflammatory |
|
4
Ligamentous strain
4
Muscle strain or spasm
4
Facet joint disruption or degeneration
4
Intervertebral disc herniation or degeneration
4
Vertebral compression fracture
4
Vertebral end-plate microfractures
4
Spondylolisthesis
4
Spinal stenosis
4
Diffuse idiopathic skeletal hyperostosis
4
Scheuermann's disease (vertebral epiphyseal aseptic necrosis) |
4
Ankylosing spondylitis
4
Reactive spondyloarthropathies (including Reiter's syndrome)
4
Psoriatic arthropathy
4
Polymyalgia rheumatica |
|
Neoplasia |
4
Epidural or vertebral carcinomatous metastasis
4
Multiple myeloma
4
Lymphoma
4
Primary epidural or intradural tumors |
|
Metabolic |
Referred pain |
4
Osteoporosis
4
Osteomalacia
4
Hemochromatosis
4
Ochronosis
4
Hyperparathyroidism |
4
Abdominal or retroperitoneal visceral process
4
Retroperitoneal vascular process
4
Retroperitoneal malignancy
4
Herpes zoster |
|
Infections |
Others |
4
Epidural abscess
4
Vertebral osteomyelitis
4
Septic discitis
4
Pott's disease
4
Nonspecific manifestation of systemic illness
4
Bacterial endocarditis
4
Influenza
|
4
Paget's disease of bone
4
Primary fibromyalgia
4
Psychogenic pain
4
Malingering
4
Emotional Stress
4
Deformities
4
Obesity |
PATHOPHYSIOLOGY
The pathophysiology
of low back pain is usually indeterminate. In fact, one of the
defining features of this disorder is its non-specific etiology.
Pain may arise from a number of sites, including the vertebral
column, surrounding muscles, tendons, ligaments, and fascia.
Stretching, tearing or contusion of these tissues may occur after
sudden unaccustomed force applied to the spine from events such as
heavy lifting, torsion of the spine, and whiplash injury.
Compression may directly stretch nociceptors (pain receptors) in
dura or nerve root sleeve tissues but ischemia from compression of
vascular structures, inflammation and secondary edema are also
likely to play a role in some cases. |
CLINICAL FEATURES
The common physical signs especially of mechanical low back pain are
local tenderness over the spine, loss of lumbar lordosis and
postural changes, lateral tilting with or without neurological
involvement. History and physical examination are critical to
diagnosis and thus to the formulation of a rational approach to
management. The following briefly summarizes the major points.
A. History
Onset of pain (e.g., time of day, activity)
Location of pain (e.g., specific site, radiation of pain)
Type and character of pain (sharp, dull, etc.)
Aggravating and relieving factors
Medical history, including previous injuries
Psychosocial stressors at home or work Red flags: age greater than
50 years, fever, weight loss
B. Physical exam
Informal observation (e.g., patient's posture, expressions, pain
behavior).
Comprehensive general physical examination, with attention to
specific areas as indicated by the history.
Neurologic evaluation.
Back examination Palpation, Range of motion or painful arc, Stance,
Gait, Mobility, Straight leg raise test.
Careful history taking and examination suffices to uncover the
mystery in most of the cases. Mechanical low back pain is
predominantly acute in onset and is frequently associated with a
history of lifting or bending. The pain may radiate to the lower
limbs and can be associated with heaviness, tingling and
paraesthesia in the thighs and legs. The pain of prolapsed lumbar
intervertebral disc (PLID) can be aggravated by coughing, sneezing
and straining during defaecation and radiates down to the back of
the leg beyond the knee. Spinal stenosis may be presented with
spinal claudication that means pain, numbness, paraesthiesia,
weakness, etc. come after standing upright or walking for 5-10
minutes and are relieved by sitting, squatting or leaning against a
wall to flex the spine. Intermittent postural low back pain, back
pain after exertion and pseudo claudication are often due to
degenerative changes like, Spondylosis, Spondylolisthesis, etc. Back
pain due to inflammatory causes are frequently associated with
prolonged early morning and inactivity stiffness. Severe and
constant back pain localized to a particular site suggests local
bone pathology, like Neoplasm, Paget's disease, compression
fracture, Pott's disease and other chronic infections, etc. Systemic
enquiry may give important clues for diagnosis of local neoplasm and
other rare causes. A healthy adult of 20-45 years are more prone to
develop PLID. Lumbar Spondylosis is related to middle age and is
common in the 5th decade. Spinal stenosis occurs commonly in people
over 50 years. Unduly protruding abdomen and peculiar stance are
often noticed by the mother of the child suffering from Dysplastic
Spondylolisthesis. Patient with chronic back pain may often develop
affective and psycho somatic ailments & their illness behavior is
often self perpetuating & self justifying. |